Antisocial Personality Disorder Essay Conclusion
Hence, it is likely that low levels of stress (E.g.
low cortisol levels and/or low heart rate) contribute to the development of ASPD in offenders.
Five studies have investigated the relationship between testosterone and antisocial behaviour in adolescents.
Several studies used the “delinquency” or “externalizing” subscales of the Child-Behaviour Checklist (CBCL) to measure antisocial behaviour. found higher levels of free testosterone in a group of high externalizing male subjects compared to a group of low externalizing male participants. used the Youth Self-Report (YSR) delinquency subscale and the Risky Behaviour Scale (RBS) to report positive correlations between free testosterone and antisocial behaviour in male subjects.
Although ASPD has a genetic basis, environmental factors greatly influence its onset.
Research has shown that the neurotransmitter serotonin (also called 5-hydroxytryptamine or 5-HT) is downregulated in ASPD patients.
Scientific evidence has found that low levels of stress contribute to the development of antisocial behaviour and ASPD.
Low levels of the neurotransmitter serotonin and maldevelopment of limbic and septal brain regions are believed to affect judgement, planning, and impulse control in ASPD sufferers.
Furthermore, environmental factors, including low socio-economic status, low education levels, and familial lifestyle, contribute to the onset of ASPD.
Decreased PFC activity was shown in antisocial criminals when compared to healthy controls.
To summarize, it is clear that hypofunctioning of the PFC contributes to the development of antisocial behaviour in criminals.